February 25, 2011

Popular heart nutrient also
stops cataracts

Cataracts are a major cause of vision loss. But you don't have to lose your vision or undergo surgery to remove cataracts. A new study suggests that one of my favorite heart nutrients can protect you.

Researchers conducted a lab study on human lens epithelial cells. They exposed the cells to white light, which is known to induce cell stress and apoptosis (death). Scientists found that preincubation of these cells with CoQ10 significantly reduced this damage. It also attenuated biochemical damage to the cells.

The scientists said cataracts are "one of the most prevalent eye diseases and a major cause of legal blindness in the world." For something so serious, these results are promising.

The weakness of this report is that they conducted it in a lab dish. However, the strength is that it does make sense. Bright light is intensely rich with energy. This energy might overcome the ability of stressed lens cells to protect themselves from that energy. CoQ10 is a critical nutrient for mitochondrial function. Mitochondria, as you may know, are your cells' energy furnaces. Cells require lots more energy to protect themselves from stress. CoQ10 might specifically lift eye-lens cells to a higher state of energy protection.

Continued Below...

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Action to take: The best way to stop cataracts is my cataract eye-drop formulation. It contains DMSO, glutathione, and vitamin C. It's not 100%. But based on feedback from users, the results are very promising. These drops do require a prescription and a compounding pharmacy to mix the solutions. You can get more information on these drops, including the best pharmacies to use, on my website.

In addition to the drops, I recommend Advanced Vision Formula. Now, I'll add CoQ10 to the list. My favorite formulation of CoQ10 is ubiquinol from Advanced Bionutritionals. One softgel twice daily should be adequate for most.

Ref: "Coenzyme Q10 prevents human lens epithelial cells from light-induced apoptotic cell death by reducing oxidative stress and stabilizing BAX / Bcl-2 ratio," Kernt M, Hirneiss C, et al, Acta Ophthalmol, 2010 April 1; [Epub ahead of print].

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