April 15, 2009

Stop liver disease with an inexpensive digestive aid

For many years, the most common cause of liver disease was alcoholic cirrhosis. But now, you're more likely than ever before to get cirrhosis — even if you don't drink. Cirrhosis has many causes, including viruses, diabetes, and obesity. But almost all of them have inflammation at their root. And once your liver is damaged, it's difficult to repair. Fortunately, there's an easy way to stop liver disease in its tracks.

A recent study found that one of my favorite digestive aids can reverse cirrhosis. In the study, researchers followed 20 patients for four weeks. All of them had alcoholic cirrhosis. The researchers gave each of them the probiotic Lactobacillus casei Shirota. The participants took the probiotic four times daily.

The researchers saw significantly improved white blood cell phagocytosis ability. That's the ability of your white blood cells to eat bacteria and foreign material. With cirrhosis, phagocytosis typically is down to 73% of normal. But with the probiotic, it recovers completely.

Probiotics should be a mainstay for most everyone in this age of toxic food. The beneficial bacteria convey a host of benefits to you. They're terrific for improving your immune system. If you have alcoholic cirrhosis, and you can find this particular strain (Lactobacillus casei Shirota), I would go with it. That's because the researchers found it effective for the specific condition. You should be able to find it at larger health food stores or on the Internet.

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For everyone else, you should take a general probiotic to prevent liver damage in the first place. I like the Advanced Probiotic Formula offered by Advanced Bionutritionals. It's one of the most powerful products on the market at a very affordable price.

Yours for better health and medical freedom,

Ref: "Effect of probiotic treatment on deranged neutrophil function and cytokine responses in patients with compensated alcoholic cirrhosis," Stadlbauer V, Mookerjee RP, et al, J Hepatol, 2008; 48(6): 945-51.

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